HPV Knowledge Challenge

Froggy Jumps

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À propos de cette activité

Test your knowledge about Human Papillomavirus with this fun quiz!

Créé par

Maria Fernanda Rodriguez

Colombia

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1	Maria Fernanda Rodriguez 4 Juin 2025	% 100	00:35

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Froggy Jumps HPV Knowledge ChallengeVersion en ligne Test your knowledge about Human Papillomavirus with this fun quiz! par Maria Fernanda Rodriguez 1 What is the main route of HPV transmission? a Sexual and skin-to-skin contact b Genetic inheritance c Blood transfusion 2 What are the high-risk HPV types most associated with cervical cancer? a HPV 6 and 11 b HPV 16 and 18 c HPV 21 and 23 3 Which viral proteins produced by high-risk HPV alter the cell cycle and can lead to cancer? a L1 y L2 b E6 y E7 c E1 y E4 4 Which cellular protein is inactivated by the viral oncoprotein E6? a p53 b p16 c pRb 5 What is the function of the late HPV genes (L1 and L2)? a Assist in the reproduction of the virus b Transform cells into cancer cells c To form the capsid of the virus 6 What happens when HPV remains latent? a It becomes a bacterium b May reactivate if immunity is lowered c It generates an immediate inflammatory response 7 Why can HPV remain in the body undetected by the immune system in the early stages? a Because it releases antibodies of its own b Because its capsid is too tough c Because it replicates in the epithelium without generating inflammation. 8 What is the consequence of the binding of E6 and E7 viral proteins to p53 and pRb in HPV-infected cells? a It enhances immune defense through antibody activation. b Induces cell differentiation and prevents viral replication c Inhibits apoptosis and allows the uncontrolled proliferation of abnormal cells

Froggy Jumps

HPV Knowledge ChallengeVersion en ligne

Test your knowledge about Human Papillomavirus with this fun quiz!

par Maria Fernanda Rodriguez

What is the main route of HPV transmission?

Sexual and skin-to-skin contact

Genetic inheritance

Blood transfusion

What are the high-risk HPV types most associated with cervical cancer?

HPV 6 and 11

HPV 16 and 18

HPV 21 and 23

Which viral proteins produced by high-risk HPV alter the cell cycle and can lead to cancer?

L1 y L2

E6 y E7

E1 y E4

Which cellular protein is inactivated by the viral oncoprotein E6?

p53

p16

pRb

What is the function of the late HPV genes (L1 and L2)?

Assist in the reproduction of the virus

Transform cells into cancer cells

To form the capsid of the virus

What happens when HPV remains latent?

It becomes a bacterium

May reactivate if immunity is lowered

It generates an immediate inflammatory response

Why can HPV remain in the body undetected by the immune system in the early stages?

Because it releases antibodies of its own

Because its capsid is too tough

Because it replicates in the epithelium without generating inflammation.

What is the consequence of the binding of E6 and E7 viral proteins to p53 and pRb in HPV-infected cells?

It enhances immune defense through antibody activation.

Induces cell differentiation and prevents viral replication

Inhibits apoptosis and allows the uncontrolled proliferation of abnormal cells

Vous pouvez intégrer le jeu dans un LMS compatible avec LTI 1.1 ou LTI 1.3 comme Canvas, Moodle ou Blackboard. Les scores seront ainsi automatiquement enregistrés dans le carnet de notes de la plateforme.

HPV Knowledge Challenge

Froggy Jumps

À propos de cette activité

Créé par

Téléchargez la version pour jouer sur papier

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Froggy Jumps

HPV Knowledge ChallengeVersion en ligne

par Maria Fernanda Rodriguez

What is the main route of HPV transmission?

What are the high-risk HPV types most associated with cervical cancer?

Which viral proteins produced by high-risk HPV alter the cell cycle and can lead to cancer?

Which cellular protein is inactivated by the viral oncoprotein E6?

What is the function of the late HPV genes (L1 and L2)?

What happens when HPV remains latent?

Why can HPV remain in the body undetected by the immune system in the early stages?

What is the consequence of the binding of E6 and E7 viral proteins to p53 and pRb in HPV-infected cells?

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