Task 5: Traumatic Brain Injury

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Answer these questions about TBI

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Kristine Vester

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Task 5: Traumatic Brain InjuryVersion en ligne Answer these questions about TBI par Kristine Vester 1 What is the primary cause of traumatic brain injury (TBI)? a An applied force on the brain. b Chemical exposure to the brain. c Natural aging of brain tissue. d Viral infections of the brain. 2 What secondary events commonly follow a primary brain injury? a Inflammation and brain edema. b Recovery of normal brain function. c Increased blood flow to the brain. d Immediate tissue repair. 3 How is the severity of a traumatic brain injury assessed? a By the Glasgow Coma Scale (GCS). b By measuring intracranial pressure directly. c By the patient's age and sex. d By the level of pain reported. 4 What is a "contrecoup injury"? a Injury on the opposite side of the brain from the initial impact. b Damage limited to the base of the skull. c Tearing of blood vessels in the brain. d Bruising only at the impact site. 5 Which of the following is a hallmark of diffuse axonal injury (DAI)? a Widespread shearing of neural networks. b Localized bleeding at the impact site. c Exclusive damage to the frontal lobe. d Gradual improvement in cognitive functions. 6 What common outcome results from increased intracranial pressure caused by TBI? a Compression of brain tissue and possible herniation. b Immediate cessation of neural activity. c Decreased oxygen levels in the bloodstream. d Total healing of brain damage. 7 What type of trauma is characterized by exposure of brain tissue to the environment? a Open blunt trauma. b Diffuse axonal injury. c Contrecoup injury. d Closed head injury. 8 Which of the following is NOT a component of the secondary injury cascade? a Increased oxygenation of brain cells. b Anoxia and hypoxemia. c Brain edema and hemorrhage. d Increased intracranial pressure. 9 What is a key challenge in diagnosing mild traumatic brain injury? a Accusations of malingering in about 30% of cases. b Inability to use imaging technology effectively. c Lack of symptoms in most patients. d Rapid recovery after injury. 10 Which research approach focuses on replacing damaged brain cells? a Stem cell transplantation. b Neuroplasticity-enhancing drugs. c Neuroprotective drug therapies. d Hormone replacement therapies. 11 What increases the risk of cumulative impairments in TBI? a Repeated mild head injuries. b Single severe head injury. c Recovery without rehabilitation. d Improved blood flow to the brain. 12 What pathology is commonly associated with the base of the skull during TBI? a Tearing and bruising of brain tissue. b Shearing of axons in the cerebellum. c Complete cessation of neural activity. d Localized edema without bleeding. 13 Which outcome is most likely with untreated mild TBI? a Persistent behavioral symptoms and cognitive impairments. b Complete recovery without intervention. c Gradual enhancement of memory functions. d Reduction in emotional instability. 14 What factor can exacerbate secondary brain injuries? a Hypotension (low blood pressure). b Increased blood clotting efficiency. c Elevated oxygen saturation levels. d High levels of physical activity. 15 What intervention shows promise for promoting neuroplasticity in TBI patients? a Physiological techniques that stimulate nerve growth. b Surgical removal of brain edema. c Long-term immobilization. d Increasing dietary protein intake. 16 What role does socioeconomic status play in TBI outcomes? a It influences access to rehabilitation and long-term care. b It determines the severity of the primary injury. c It directly impacts the extent of neural damage. d It dictates the speed of recovery. 17 Which factor can prevent herniation in TBI patients? a Early control of intracranial pressure. b Avoiding physical therapy. c Delaying surgical intervention. d Enhanced hydration therapy. 18 Which brain function is commonly impaired after TBI? a Short-term memory processing. b Long-term vision clarity. c Enhanced motor control. d Increased auditory sensitivity. 19 What is the primary limitation of current TBI treatments? a Lack of clinically effective methods to repair brain damage. b Excessive reliance on stem cell research. c Overuse of neuroprotective drugs. d Lack of diagnostic imaging technology. 20 What symptom is most characteristic of diffuse axonal injury? a Widespread neuronal death. b Isolated bruising at the impact site. c Enhanced blood flow in the brain. d Immediate full recovery.

Task 5: Traumatic Brain InjuryVersion en ligne

Answer these questions about TBI

par Kristine Vester

What is the primary cause of traumatic brain injury (TBI)?

An applied force on the brain.

Chemical exposure to the brain.

Natural aging of brain tissue.

Viral infections of the brain.

What secondary events commonly follow a primary brain injury?

Inflammation and brain edema.

Recovery of normal brain function.

Increased blood flow to the brain.

Immediate tissue repair.

How is the severity of a traumatic brain injury assessed?

By the Glasgow Coma Scale (GCS).

By measuring intracranial pressure directly.

By the patient's age and sex.

By the level of pain reported.

What is a "contrecoup injury"?

Injury on the opposite side of the brain from the initial impact.

Damage limited to the base of the skull.

Tearing of blood vessels in the brain.

Bruising only at the impact site.

Which of the following is a hallmark of diffuse axonal injury (DAI)?

Widespread shearing of neural networks.

Localized bleeding at the impact site.

Exclusive damage to the frontal lobe.

Gradual improvement in cognitive functions.

What common outcome results from increased intracranial pressure caused by TBI?

Compression of brain tissue and possible herniation.

Immediate cessation of neural activity.

Decreased oxygen levels in the bloodstream.

Total healing of brain damage.

What type of trauma is characterized by exposure of brain tissue to the environment?

Open blunt trauma.

Diffuse axonal injury.

Contrecoup injury.

Closed head injury.

Which of the following is NOT a component of the secondary injury cascade?

Increased oxygenation of brain cells.

Anoxia and hypoxemia.

Brain edema and hemorrhage.

Increased intracranial pressure.

What is a key challenge in diagnosing mild traumatic brain injury?

Accusations of malingering in about 30% of cases.

Inability to use imaging technology effectively.

Lack of symptoms in most patients.

Rapid recovery after injury.

Which research approach focuses on replacing damaged brain cells?

Stem cell transplantation.

Neuroplasticity-enhancing drugs.

Neuroprotective drug therapies.

Hormone replacement therapies.

What increases the risk of cumulative impairments in TBI?

Repeated mild head injuries.

Single severe head injury.

Recovery without rehabilitation.

Improved blood flow to the brain.

What pathology is commonly associated with the base of the skull during TBI?

Tearing and bruising of brain tissue.

Shearing of axons in the cerebellum.

Complete cessation of neural activity.

Localized edema without bleeding.

Which outcome is most likely with untreated mild TBI?

Persistent behavioral symptoms and cognitive impairments.

Complete recovery without intervention.

Gradual enhancement of memory functions.

Reduction in emotional instability.

What factor can exacerbate secondary brain injuries?

Hypotension (low blood pressure).

Increased blood clotting efficiency.

Elevated oxygen saturation levels.

High levels of physical activity.

What intervention shows promise for promoting neuroplasticity in TBI patients?

Physiological techniques that stimulate nerve growth.

Surgical removal of brain edema.

Long-term immobilization.

Increasing dietary protein intake.

What role does socioeconomic status play in TBI outcomes?

It influences access to rehabilitation and long-term care.

It determines the severity of the primary injury.

It directly impacts the extent of neural damage.

It dictates the speed of recovery.

Which factor can prevent herniation in TBI patients?

Early control of intracranial pressure.

Avoiding physical therapy.

Delaying surgical intervention.

Enhanced hydration therapy.

Which brain function is commonly impaired after TBI?

Short-term memory processing.

Long-term vision clarity.

Enhanced motor control.

Increased auditory sensitivity.

What is the primary limitation of current TBI treatments?

Lack of clinically effective methods to repair brain damage.

Excessive reliance on stem cell research.

Overuse of neuroprotective drugs.

Lack of diagnostic imaging technology.

What symptom is most characteristic of diffuse axonal injury?

Widespread neuronal death.

Isolated bruising at the impact site.

Enhanced blood flow in the brain.

Immediate full recovery.