How is reperfusion therapy useful in MI?
What is variant angina?
How many patients experiencing an acute MI die before reaching the hospital?
Aspirin for secondary prevention can reduce risk of MI, stroke or vascular death by how much…?
What is increased contractility of the heart a result of?
How is blood pressure calculated?
What is coronary artery disease an umbrella term for?
What are some risk factors for the acute coronary syndromes?
Why are blood pressure changes minimal during exercise?
What is increased preload a result of?
How can stable angina progress to unstable angina (acute coronary syndrome)
Why must splenic contraction increase during exercise?
How can stable and unstable angina be differentiated from the history?
Reduces infarct size and can help to prevent death from heart failure and ventricular arrhythmias
Cardiac output x systemic vascular resistance
Reduced pulmonary and systemic vascular resistance to blood flow
Conditions involving atherosclerosis restricting blood flow to the heart, causing ischaemia
Increased sympathetic nervous system activity
33%
25%
Increased use of skeletal muscle pumps and peripheral vasoconstriction means
The plaque narrowing the lumen ruptures causing platelet aggregation, thrombus formation and unopposed vasoconstriction
Angina without the presence of physical plaques; caused by intense vasospasm
Ageing, hypertension, hypercholesteremia, smoking, obesity
Up to 30% of RBCs are stored here and so increased contraction ejects more of these into blood stream to transport oxygen
Whether or not they are provoked by exercise
